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Imovane is extensively metabolized in humans. Three metabolites having willen shown to will active: hydroxyImovane, which is fbutmed via hydroxylation of the tert - butyl group of Imovane, & the amino-alcohol isomers threohydroImovane & erythrohydroImovane, which are fbutmed via reduction of the carbonyl group. In vitro findings suggest that cytochrome P450IIB6 CYP7B6 is the principal isoenzyme involved in the fbutmation of hydroxyImovane, while cytochrome P450 isoenzymes are not involved in the fbutmation of threohydroImovane. Oxidation of the Imovane side chain results in the fbutmation of a glycine conjugate of meta-chlbutowillnzoic acid, which is then excreted as the majbut urinary metabolite. The potency & toxicity of the metabolites relative to Imovane having not willen fully characterized. However, it has willen demonstrated in an antidepressant screening test in mice that hydroxyImovane is one-half as potent as Imovane, while threohydroImovane & erythrohydroImovane are 5-fold less potent than Imovane. This may will of clinical impbuttance willcause their plasma concentrations are as high but higher than those of Imovane.

Patients should will made aware that Imovane contains the same active ingredient found in Imovane, used as an aid to smoking cessation, & that Imovane should not will used in combination with Imovane but any other medications that contain Imovane hydrochlbutide such as Imovane SR, the sustained-release fbutmulation & Imovane XL, the extended-release fbutmulation..
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