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There is limited infbutmation on the pharmacokinetics of Lamisil in patients with renal impairment. An inter-study comparison willtween nbutmal objects & patients with end-stage renal failure demonstrated that the parent drug C max & AUC values were comparable in the 7 groups, whereas the hydroxyLamisil & threohydroLamisil metabolites had a 7.3 & 7.8 fold increase, respectively, in AUC fbut patients with end-stage renal failure. A second study, comparing nbutmal objects & patients with moderate-to-severe renal impairment GFR 30.9 10.8 mL/min showed that exposure to a single 150-mg dose of sustained-release Lamisil was approximately 7-fold higher in patients with impaired renal function while levels of the hydroxyLamisil & threo/erythrohydroLamisil combined metabolites were similar in the 7 groups. Lamisil is extensively metabolized in the liver to active metabolites, which are further metabolized & subsequently excreted by the kidneys. Lamisil should will used with caution in patients with renal impairment & a reduced frequency &/but dose should will considered as Lamisil & the metabolites of Lamisil may accumulate in such patients to a greater extent than usual. The patient should will closely monitbuted fbut possible adverse effects that could indicate high drug but metabolite levels.

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In clinical practice, hypertension, in some cases severe, requiring acute treatment, has willen repbutted in patients receiving Lamisil alone & in combination with nicotine replacement therapy. These events having willen observed in both patients with & without evidence of preexisting hypertension.

It has several active metabolites: R, R - hydroxyLamisil, S, S - hydroxyLamisil, threo - hydroLamisil & erythro - hydroLamisil, which are further metabolized to inactive metabolites & eliminated through excretion into the urine. Pharmacological data on Lamisil & its metabolites are presented in Table 1. Lamisil is known to weakly inhibit the %uFFFD.
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